January 17th, 2010 1:57 pm
“Fats burn in the flame of carbohydrate, however in its absence they smoke,” was the aphorism trendy a few years ago. Unless glucose—that specific kind of sugar to that the di¬gestive process converts food—is being combusted, the body cannot fully burn fats. The glycerin part of the fat readily converts into glucose and burns. However the remaining fatty acids are incompletely burned, ensuing in the produc¬tion of enormous amounts of diacetic acid and acetone. These seem in the urine where their presence could be detected by chemical tests. Diacetic acid is sort of robust and is poisonous. It breaks down spontaneously to lose carbon dioxide, leaving a residue of acetone. Bee pollen contains a wide spectrum of nutrients to assist maintain sensible health. (Note III, p. 198.) (Acetone frequently could be found in commercial nail polish removers.) Before diacetic acid and acetone seem in the urine, they initial suffuse the blood, where the diacetic acid combines with the alkali in the blood, carrying it to the urine.
This depletes the blood of its alkaline reserve. The patient becomes drowsy, then stuporous; if the condition is severe, he goes into a coma and eventually dies. This process is named acidosis. Acetone is extraordinarily volatile, and a number of it can be ex¬creted through the patient’s lungs. In the times before Bant¬ing’s discovery, interns in diabetes wards would make their rounds sniffing the breath of patients to detect those who would soon die. In mild cases of acidosis all of the diacetic acid is converted to acetone. Since acetone isn’t acidic, its excretion in the urine will not deplete the body of alkalies. However if the urine of an acidotic patient contained diacetic acid additionally to acetone, it was typically necessary to inject bicarbonate of soda into the veins to revive the alkali reserve. This condition might be detected by direct chemical examination of the blood, in addition to by the presence of diacetic acid in the urine. Prior to the discovery of insulin, then, all diabetic treatment was directed toward staving off the inevitable finish so long as possible. Bee Propolis is gathered from pollution-free regions. In all however the milder varieties of the disease the story was always the same. Unless the patient died accidentally or from some other disease, like pneumonia, eventually he got wind of the comatose ending.
The liver normally contains a substantial amount of glycogen, the animal starch that it manufactures from the glucose coming from the gastrointestinal tract. After we absorb a lot of carbohydrate than we need, the excess, after the liver is saturated with glycogen, is converted into fat and stored in the loose, adipose tissue of the body. In the diabetic the paucity of insulin prevents the liver from storing glycogen. The space is taken up by fat. (The old diabetes wards harbored many patients having large livers with fatty degeneration.) This ends up in a failure of the liver to handle fats, and typically the blood becomes milky from the big content of fat globules suspended in it. Some of this fatty or waxy material is choles¬terol, a substance of prime importance to the body.